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Items where Author is "Geiszt, Miklós"

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Number of items: 7.

Article

Margittai, Éva and Enyedi, Balázs and Csala, Miklós and Geiszt, Miklós and Bánhegyi, Gábor (2015) Composition of the redox environment of the endoplasmic reticulum and sources of hydrogen peroxide. FREE RADICAL BIOLOGY AND MEDICINE, 83. pp. 331-340. ISSN 0891-5849

Hristova, Milena and Veith, Carmen and Habibovic, Aida and Lam, Ying-Wai and Deng, Bin and Geiszt, Miklós (2014) Identification of DUOX1-dependent redox signaling through protein S-glutathionylation in airway epithelial cells. REDOX BIOLOGY, 2 (1). pp. 436-446. ISSN 2213-2317, ESSN: 2213-2317

Pape, Veronika F. S. and Enyedy, Éva Anna and Lovrics, Anna and Nagy, Zsuzsanna and Kucsma, Nóra and Szepesi, Áron and Geiszt, Miklós and Szakács, Gergely (2014) Investigating the impact of ROS on the toxicity of plumbagin and its copper complexes. JOURNAL OF BIOLOGICAL INORGANIC CHEMISTRY, 19 (2). S879-S879. ISSN 0949-8257

Fekete, Anna and Bőgel, Gábor and Pesti, Szabolcs and Péterfi, Zalán and Geiszt, Miklós and Buday, László (2013) EGF regulates tyrosine phosphorylation and membrane-translocation of the scaffold protein Tks5. JOURNAL OF MOLECULAR SIGNALING, 8. pp. 1-8. ISSN 1750-2187

Monograph

Geiszt, Miklós and Donkó, Ágnes and Enyedi, Balázs and Lányi, Árpád and Orient, Anna and Péterfi, Zalán (2012) Reaktív oxigén származékok szerepe a fibrózis kialakulásában = Reactive oxygen species in the development of organ fibrosis. Project Report. OTKA.

Geiszt, Miklós and Hably, Csilla and Rada, Balázs and Vág, János and Várnai, Péter (2007) A Renox szuperoxid-termelő enzim vizsgálata emlős sejtekben = Characterization of the Renox superoxide-producing enzyme in mammalian cells. Project Report. OTKA.

Ligeti, Erzsébet and Geiszt, Miklós and Káldi, Krisztina and Molnár, Gergely and Patryk, Moskwa and Rada, Balázs (2006) NADPH oxidáz szabályozása és élettani szerepe = Regulation and physiological role of NADPH oxidase. Project Report. OTKA.

This list was generated on Mon Aug 31 08:45:18 2015 CEST.